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This change in available IGF-1 may be (it is difficult to extrapolate data from altered IGF-1/GH concentrations from animal models, as each author measures IGF-1 levels using different methods) adequate for fatty acid (FA) oxidation in muscle, suppression of GH, stimulation of glucose transport into muscle [171, 172], and lastly for the suppression of renal gluconeogenesis in mice [163]

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Although the total concentrations (oxidized plus reduced forms) of NAD(H) and NADP(H) in the RBC are similar (~25 mol/L), the cell maintains these two redox systems with similar redox potentials at such different set-points in the same cell by isolating their metabolism through the specificity of cytoplasmic dehydrogenases

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For cardiovascular and metabolic benefits, longer treatment durations produce more sustained protection